Evolution is slow–until it’s fast: Genetic Load and the Future of Humanity

Source: Priceonomics

A species may live in relative equilibrium with its environment, hardly changing from generation to generation, for millions of years. Turtles, for example, have barely changed since the Cretaceous, when dinosaurs still roamed the Earth.

But if the environment changes–critically, if selective pressures change–then the species will change, too. This was most famously demonstrated with English moths, which changed color from white-and-black speckled to pure black when pollution darkened the trunks of the trees they lived on. To survive, these moths need to avoid being eaten by birds, so any moth that stands out against the tree trunks tends to get turned into an avian snack. Against light-colored trees, dark-colored moths stood out and were eaten. Against dark-colored trees, light-colored moths stand out.

This change did not require millions of years. Dark-colored moths were virtually unknown in 1810, but by 1895, 98% of the moths were black.

The time it takes for evolution to occur depends simply on A. The frequency of a trait in the population and B. How strongly you are selecting for (or against) it.

Let’s break this down a little bit. Within a species, there exists a great deal of genetic variation. Some of this variation happens because two parents with different genes get together and produce offspring with a combination of their genes. Some of this variation happens because of random errors–mutations–that occur during copying of the genetic code. Much of the “natural variation” we see today started as some kind of error that proved to be useful, or at least not harmful. For example, all humans originally had dark skin similar to modern Africans’, but random mutations in some of the folks who no longer lived in Africa gave them lighter skin, eventually producing “white” and “Asian” skin tones.

(These random mutations also happen in Africa, but there they are harmful and so don’t stick around.)

Natural selection can only act on the traits that are actually present in the population. If we tried to select for “ability to shoot x-ray lasers from our eyes,” we wouldn’t get very far, because no one actually has that mutation. By contrast, albinism is rare, but it definitely exists, and if for some reason we wanted to select for it, we certainly could. (The incidence of albinism among the Hopi Indians is high enough–1 in 200 Hopis vs. 1 in 20,000 Europeans generally and 1 in 30,000 Southern Europeans–for scientists to discuss whether the Hopi have been actively selecting for albinism. This still isn’t a lot of albinism, but since the American Southwest is not a good environment for pale skin, it’s something.)

You will have a much easier time selecting for traits that crop up more frequently in your population than traits that crop up rarely (or never).

Second, we have intensity–and variety–of selective pressure. What % of your population is getting removed by natural selection each year? If 50% of your moths get eaten by birds because they’re too light, you’ll get a much faster change than if only 10% of moths get eaten.

Selection doesn’t have to involve getting eaten, though. Perhaps some of your moths are moth Lotharios, seducing all of the moth ladies with their fuzzy antennae. Over time, the moth population will develop fuzzier antennae as these handsome males out-reproduce their less hirsute cousins.

No matter what kind of selection you have, nor what part of your curve it’s working on, all that ultimately matters is how many offspring each individual has. If white moths have more children than black moths, then you end up with more white moths. If black moths have more babies, then you get more black moths.

Source SUPS.org

So what happens when you completely remove selective pressures from a population?

Back in 1968, ethologist John B. Calhoun set up an experiment popularly called “Mouse Utopia.” Four pairs of mice were given a large, comfortable habitat with no predators and plenty of food and water.

Predictably, the mouse population increased rapidly–once the mice were established in their new homes, their population doubled every 55 days. But after 211 days of explosive growth, reproduction began–mysteriously–to slow. For the next 245 days, the mouse population doubled only once every 145 days.

The birth rate continued to decline. As births and death reached parity, the mouse population stopped growing. Finally the last breeding female died, and the whole colony went extinct.

source

As I’ve mentioned before Israel is (AFAIK) the only developed country in the world with a TFR above replacement.

It has long been known that overcrowding leads to population stress and reduced reproduction, but overcrowding can only explain why the mouse population began to shrink–not why it died out. Surely by the time there were only a few breeding pairs left, things had become comfortable enough for the remaining mice to resume reproducing. Why did the population not stabilize at some comfortable level?

Professor Bruce Charlton suggests an alternative explanation: the removal of selective pressures on the mouse population resulted in increasing mutational load, until the entire population became too mutated to reproduce.

What is genetic load?

As I mentioned before, every time a cell replicates, a certain number of errors–mutations–occur. Occasionally these mutations are useful, but the vast majority of them are not. About 30-50% of pregnancies end in miscarriage (the percent of miscarriages people recognize is lower because embryos often miscarry before causing any overt signs of pregnancy,) and the majority of those miscarriages are caused by genetic errors.

Unfortunately, randomly changing part of your genetic code is more likely to give you no skin than skintanium armor.

But only the worst genetic problems that never see the light of day. Plenty of mutations merely reduce fitness without actually killing you. Down Syndrome, famously, is caused by an extra copy of chromosome 21.

While a few traits–such as sex or eye color–can be simply modeled as influenced by only one or two genes, many traits–such as height or IQ–appear to be influenced by hundreds or thousands of genes:

Differences in human height is 60–80% heritable, according to several twin studies[19] and has been considered polygenic since the Mendelian-biometrician debate a hundred years ago. A genome-wide association (GWA) study of more than 180,000 individuals has identified hundreds of genetic variants in at least 180 loci associated with adult human height.[20] The number of individuals has since been expanded to 253,288 individuals and the number of genetic variants identified is 697 in 423 genetic loci.[21]

Obviously most of these genes each plays only a small role in determining overall height (and this is of course holding environmental factors constant.) There are a few extreme conditions–gigantism and dwarfism–that are caused by single mutations, but the vast majority of height variation is caused by which particular mix of those 700 or so variants you happen to have.

The situation with IQ is similar:

Intelligence in the normal range is a polygenic trait, meaning it’s influenced by more than one gene.[3][4]

The general figure for the heritability of IQ, according to an authoritative American Psychological Association report, is 0.45 for children, and rises to around 0.75 for late teens and adults.[5][6] In simpler terms, IQ goes from being weakly correlated with genetics, for children, to being strongly correlated with genetics for late teens and adults. … Recent studies suggest that family and parenting characteristics are not significant contributors to variation in IQ scores;[8] however, poor prenatal environment, malnutrition and disease can have deleterious effects.[9][10]

And from a recent article published in Nature Genetics, Genome-wide association meta-analysis of 78,308 individuals identifies new loci and genes influencing human intelligence:

Despite intelligence having substantial heritability2 (0.54) and a confirmed polygenic nature, initial genetic studies were mostly underpowered3, 4, 5. Here we report a meta-analysis for intelligence of 78,308 individuals. We identify 336 associated SNPs (METAL P < 5 × 10−8) in 18 genomic loci, of which 15 are new. Around half of the SNPs are located inside a gene, implicating 22 genes, of which 11 are new findings. Gene-based analyses identified an additional 30 genes (MAGMA P < 2.73 × 10−6), of which all but one had not been implicated previously. We show that the identified genes are predominantly expressed in brain tissue, and pathway analysis indicates the involvement of genes regulating cell development (MAGMA competitive P = 3.5 × 10−6). Despite the well-known difference in twin-based heritability2 for intelligence in childhood (0.45) and adulthood (0.80), we show substantial genetic correlation (rg = 0.89, LD score regression P = 5.4 × 10−29). These findings provide new insight into the genetic architecture of intelligence.

The greater number of genes influence a trait, the harder they are to identify without extremely large studies, because any small group of people might not even have the same set of relevant genes.

High IQ correlates positively with a number of life outcomes, like health and longevity, while low IQ correlates with negative outcomes like disease, mental illness, and early death. Obviously this is in part because dumb people are more likely to make dumb choices which lead to death or disease, but IQ also correlates with choice-free matters like height and your ability to quickly press a button. Our brains are not some mysterious entities floating in a void, but physical parts of our bodies, and anything that affects our overall health and physical functioning is likely to also have an effect on our brains.

Like height, most of the genetic variation in IQ is the combined result of many genes. We’ve definitely found some mutations that result in abnormally low IQ, but so far we have yet (AFAIK) to find any genes that produce the IQ gigantism. In other words, low (genetic) IQ is caused by genetic load–Small Yet Important Genetic Differences Between Highly Intelligent People and General Population:

The study focused, for the first time, on rare, functional SNPs – rare because previous research had only considered common SNPs and functional because these are SNPs that are likely to cause differences in the creation of proteins.

The researchers did not find any individual protein-altering SNPs that met strict criteria for differences between the high-intelligence group and the control group. However, for SNPs that showed some difference between the groups, the rare allele was less frequently observed in the high intelligence group. This observation is consistent with research indicating that rare functional alleles are more often detrimental than beneficial to intelligence.

Maternal mortality rates over time, UK data

Greg Cochran has some interesting Thoughts on Genetic Load. (Currently, the most interesting candidate genes for potentially increasing IQ also have terrible side effects, like autism, Tay Sachs and Torsion Dystonia. The idea is that–perhaps–if you have only a few genes related to the condition, you get an IQ boost, but if you have too many, you get screwed.) Of course, even conventional high-IQ has a cost: increased maternal mortality (larger heads).

Wikipedia defines genetic load as:

the difference between the fitness of an average genotype in a population and the fitness of some reference genotype, which may be either the best present in a population, or may be the theoretically optimal genotype. … Deleterious mutation load is the main contributing factor to genetic load overall.[5] Most mutations are deleterious, and occur at a high rate.

There’s math, if you want it.

Normally, genetic mutations are removed from the population at a rate determined by how bad they are. Really bad mutations kill you instantly, and so are never born. Slightly less bad mutations might survive, but never reproduce. Mutations that are only a little bit deleterious might have no obvious effect, but result in having slightly fewer children than your neighbors. Over many generations, this mutation will eventually disappear.

(Some mutations are more complicated–sickle cell, for example, is protective against malaria if you have only one copy of the mutation, but gives you sickle cell anemia if you have two.)

Jakubany is a town in the Carpathian Mountains

Throughout history, infant mortality was our single biggest killer. For example, here is some data from Jakubany, a town in the Carpathian Mountains:

We can see that, prior to the 1900s, the town’s infant mortality rate stayed consistently above 20%, and often peaked near 80%.

The graph’s creator states:

When I first ran a calculation of the infant mortality rate, I could not believe certain of the intermediate results. I recompiled all of the data and recalculated … with the same astounding result – 50.4% of the children born in Jakubany between the years 1772 and 1890 would diebefore reaching ten years of age! …one out of every two! Further, over the same 118 year period, of the 13306 children who were born, 2958 died (~22 %) before reaching the age of one.

Historical infant mortality rates can be difficult to calculate in part because they were so high, people didn’t always bother to record infant deaths. And since infants are small and their bones delicate, their burials are not as easy to find as adults’. Nevertheless, Wikipedia estimates that Paleolithic man had an average life expectancy of 33 years:

Based on the data from recent hunter-gatherer populations, it is estimated that at 15, life expectancy was an additional 39 years (total 54), with a 0.60 probability of reaching 15.[12]

Priceonomics: Why life expectancy is misleading

In other words, a 40% chance of dying in childhood. (Not exactly the same as infant mortality, but close.)

Wikipedia gives similarly dismal stats for life expectancy in the Neolithic (20-33), Bronze and Iron ages (26), Classical Greece(28 or 25), Classical Rome (20-30), Pre-Columbian Southwest US (25-30), Medieval Islamic Caliphate (35), Late Medieval English Peerage (30), early modern England (33-40), and the whole world in 1900 (31).

Over at ThoughtCo: Surviving Infancy in the Middle Ages, the author reports estimates for between 30 and 50% infant mortality rates. I recall a study on Anasazi nutrition which I sadly can’t locate right now, which found 100% malnutrition rates among adults (based on enamel hypoplasias,) and 50% infant mortality.

As Priceonomics notes, the main driver of increasing global life expectancy–48 years in 1950 and 71.5 years in 2014 (according to Wikipedia)–has been a massive decrease in infant mortality. The average life expectancy of an American newborn back in 1900 was only 47 and a half years, whereas a 60 year old could expect to live to be 75. In 1998, the average infant could expect to live to about 75, and the average 60 year old could expect to live to about 80.

Back in his post on Mousetopia, Charlton writes:

Michael A Woodley suggests that what was going on [in the Mouse experiment] was much more likely to be mutation accumulation; with deleterious (but non-fatal) genes incrementally accumulating with each generation and generating a wide range of increasingly maladaptive behavioural pathologies; this process rapidly overwhelming and destroying the population before any beneficial mutations could emerge to ‘save; the colony from extinction. …

The reason why mouse utopia might produce so rapid and extreme a mutation accumulation is that wild mice naturally suffer very high mortality rates from predation. …

Thus mutation selection balance is in operation among wild mice, with very high mortality rates continually weeding-out the high rate of spontaneously-occurring new mutations (especially among males) – with typically only a small and relatively mutation-free proportion of the (large numbers of) offspring surviving to reproduce; and a minority of the most active and healthy (mutation free) males siring the bulk of each generation.

However, in Mouse Utopia, there is no predation and all the other causes of mortality (eg. Starvation, violence from other mice) are reduced to a minimum – so the frequent mutations just accumulate, generation upon generation – randomly producing all sorts of pathological (maladaptive) behaviours.

Historically speaking, another selective factor operated on humans: while about 67% of women reproduced, only 33% of men did. By contrast, according to Psychology Today, a majority of today’s men have or will have children.

Today, almost everyone in the developed world has plenty of food, a comfortable home, and doesn’t have to worry about dying of bubonic plague. We live in humantopia, where the biggest factor influencing how many kids you have is how many you want to have.

source

Back in 1930, infant mortality rates were highest among the children of unskilled manual laborers, and lowest among the children of professionals (IIRC, this is Brittish data.) Today, infant mortality is almost non-existent, but voluntary childlessness has now inverted this phenomena:

Yes, the percent of childless women appears to have declined since 1994, but the overall pattern of who is having children still holds. Further, while only 8% of women with post graduate degrees have 4 or more children, 26% of those who never graduated from highschool have 4+ kids. Meanwhile, the age of first-time moms has continued to climb.

In other words, the strongest remover of genetic load–infant mortality–has all but disappeared; populations with higher load (lower IQ) are having more children than populations with lower load; and everyone is having children later, which also increases genetic load.

Take a moment to consider the high-infant mortality situation: an average couple has a dozen children. Four of them, by random good luck, inherit a good combination of the couple’s genes and turn out healthy and smart. Four, by random bad luck, get a less lucky combination of genes and turn out not particularly healthy or smart. And four, by very bad luck, get some unpleasant mutations that render them quite unhealthy and rather dull.

Infant mortality claims half their children, taking the least healthy. They are left with 4 bright children and 2 moderately intelligent children. The three brightest children succeed at life, marry well, and end up with several healthy, surviving children of their own, while the moderately intelligent do okay and end up with a couple of children.

On average, society’s overall health and IQ should hold steady or even increase over time, depending on how strong the selective pressures actually are.

Or consider a consanguineous couple with a high risk of genetic birth defects: perhaps a full 80% of their children die, but 20% turn out healthy and survive.

Today, by contrast, your average couple has two children. One of them is lucky, healthy, and smart. The other is unlucky, unhealthy, and dumb. Both survive. The lucky kid goes to college, majors in underwater intersectionist basket-weaving, and has one kid at age 40. That kid has Down Syndrome and never reproduces. The unlucky kid can’t keep a job, has chronic health problems, and 3 children by three different partners.

Your consanguineous couple migrates from war-torn Somalia to Minnesota. They still have 12 kids, but three of them are autistic with IQs below the official retardation threshold. “We never had this back in Somalia,” they cry. “We don’t even have a word for it.”

People normally think of dysgenics as merely “the dumb outbreed the smart,” but genetic load applies to everyone–men and women, smart and dull, black and white, young and especially old–because we all make random transcription errors when copying our DNA.

I could offer a list of signs of increasing genetic load, but there’s no way to avoid cherry-picking trends I already know are happening, like falling sperm counts or rising (diagnosed) autism rates, so I’ll skip that. You may substitute your own list of “obvious signs society is falling apart at the genes” if you so desire.

Nevertheless, the transition from 30% (or greater) infant mortality to almost 0% is amazing, both on a technical level and because it heralds an unprecedented era in human evolution. The selective pressures on today’s people are massively different from those our ancestors faced, simply because our ancestors’ biggest filter was infant mortality. Unless infant mortality acted completely at random–taking the genetically loaded and unloaded alike–or on factors completely irrelevant to load, the elimination of infant mortality must continuously increase the genetic load in the human population. Over time, if that load is not selected out–say, through more people being too unhealthy to reproduce–then we will end up with an increasing population of physically sick, maladjusted, mentally ill, and low-IQ people.

(Remember, all mental traits are heritable–so genetic load influences everything, not just controversial ones like IQ.)

If all of the above is correct, then I see only 4 ways out:

  1. Do nothing: Genetic load increases until the population is non-functional and collapses, resulting in a return of Malthusian conditions, invasion by stronger neighbors, or extinction.
  2. Sterilization or other weeding out of high-load people, coupled with higher fertility by low-load people
  3. Abortion of high load fetuses
  4. Genetic engineering

#1 sounds unpleasant, and #2 would result in masses of unhappy people. We don’t have the technology for #4, yet. I don’t think the technology is quite there for #2, either, but it’s much closer–we can certainly test for many of the deleterious mutations that we do know of.

Open Thread: Education and Survival

Really dumb people are too dumb to commit as much crime as mildly dumb people
Graph of IQ vs crime — Really dumb people are too dumb to commit as much crime as mildly dumb people

Hey guys, I have a cold. Boo. How are you?

Today’s theme is education. You probably know already that I’m pretty positive toward both public/private schools and homeschooling; I think which you should chose depends a lot on a person/family’s individual situation.

But what could we do to improve these systems? (Imagine you are given free-range to design a system from scratch.) What would you add or subtract? Would you change the focus or style in some way? (Do you have any specific recommendations for books or curriculum materials for children?)

One thing I find lacking in the modern school system is a clear path to a job. A highschool diploma ought to qualify a person for many low-level jobs, but as a practical matter, it’s basically crap. A college degree ought to qualify you for the average higher-skill job, but even still, there’s a big disconnect between getting the degree and getting the job. I’ve known people with degrees from very nice schools (HYPS-MC) who have still struggled to get good, regular employment. And many people end up working in fields well outside of what they majored in. That’s not horrible–life happens–but it does make me question what the whole point of spending 4 years and $$$ on a degree in the first place was. (If it’s signaling, we could do signaling a lot cheaper.)

Scheme of the Roman Hierapolis sawmill, the earliest known machine to incorporate a crank and connecting rod mechanism.
Scheme of the Roman Hierapolis sawmill, the earliest known machine to incorporate a crank and connecting rod mechanism.

Anyway, I was thinking about survival as a skill, man-vs-wild style. How to hunt/fish/trap/gather your food. How to build a shelter. How to signal SOS. Basic woodworking? First aid, navigation, swimming, boating. What would you add?

Relatedly: how to start a business and actually make money. How to fill out the necessary related forms.

How would you go about teaching that/finding people to teach it? We’re in cub/girl scouts, but I find those basically useless; I don’t think my kids have learned so much as to tie a knot there in the past 3 years. (Don’t get me wrong, they’re still having fun. They’re just not about to come home with freshly killed dinner anytime soon.)

I hate the phrase "Red pill" because it's dumb, but something similar to this was my red pill moment. This isn't about justice; it's about being mad that they aren't allowed to murder us without consequences.
I hate the phrase “Red pill” because it’s dumb, but something similar to this was my red pill moment. This isn’t about justice; it’s about being mad that they aren’t allowed to murder us without consequences.

I hear people say, “kids are natural learners, we shouldn’t force them to learn!” Well my kids are naturals at learning Minecraft, but they think multiplication is lame.

So I’m eager to hear your thoughts.

Some interesting Links:

Walruses try to play with seabirds. No word yet on if seabirds play back:

Behavioural analysis based on detailed observations, photography and video recording showed that the most common types of walrus behaviour toward a bird were approach by surfacing and splash, approach by surfacing and hit and attack from below. Immature individuals initiated 82% of encounters. … Walrus encounters with live birds showed a very low rate of bird kill. … Object play in wild walruses is reported for the first time.

The porbeagle shark plays with seaweed:

This stocky shark is often included in studies on whether or not sharks play. That is because several observers have reported seeing porbeagles in groups of up to 20 individuals manipulating and tossing about floating objects, including lumber and seaweed. They seem to engage in such activity for no apparent reason other than to pass the time.

Okay, comments o the week:

Leuconoe brings up an argument I hadn’t even thought of before regarding corn, potatoes, and exchange with the New World:

The effects of the exchange were various, on the one hand it brought deadly illneses to the new world that killed tens of millions on the other it brought many forms of food to bouth worlds that saved hundreds of millions from starvation. Chinas population grew from 150 to 400 million because of introduction of new world crops. What would have hapened to this people without the crops? They would have died from malthusian limits or killed by their parents.

There were many other excellent comments, but I think I will highlight infowarrior1‘s question and follow-up (after I said it seemed inefficient):

I have a question. Do you regard war as eugenic or dysgenic as it currently stands?

Then why are humans and chimpanzees designed to war in the 1st place if its so inefficient?

Discuss!

 

Re: Chauncey Tinker on Dysgenics

By request, I am responding to Chauncey Tinker’s posts on dysgenics:

Dysgenics and Welfare and Dysgenics and Solutions.

To summarize, our current generous welfare system is making it increasingly difficult for hard working members of society to afford to have children. Lazy and incapable people meanwhile are continuing to have children without restriction, courtesy of those hard working people. Its more than likely that average intelligence is falling as a result of these pressures.

Ever since someone proposed the idea of eguenic (ie, good) breeding, people have been concerned by the possibility of dysgenic (bad) breeding. If traits are heritable (as, indeed, they are,) then you can breed for more of that trait or less of that trait. Anyone who has ever raised livestock or puppies knows as much–the past 10,000 years of animal husbandry have been devoted to producing superior stock, long before anyone knew anything about “genes.”

Historically–that is, before 1900–the world was harsh and survival far from guaranteed. Infant and childhood mortality were high, women often died in childbirth, famines were frequent, land (in Europe) was scarce, and warfare + polygamy probably prevented the majority of men from ever reproducing. In those days, at least in Western Europe, the upper classes tended to have more (surviving) children than the lower classes, leading to a gradual replacement of the lower classes.

The situation today is, obviously, radically different. Diseases–genetic or pathogenic–kill far fewer people. We can cure Bubonic Plague with penicillin, have wiped out Smallpox, and can perform heart surgery on newborns whose hearts were improperly formed. Welfare prevents people from starving in the streets and the post-WWII prosperity led to an unprecedented percent of men marrying and raising families. (The percent of women who married and raised families probably didn’t change that much.)

All of these pleasant events raise concerns that, long-term, prosperity could result in the survival of people whose immune systems are weak, carry rare but debilitating genetic mutations, or are just plain dumb.

So how is Western fertility? Are the dumb outbreeding the smart, or should we be grateful that the  “gender studies” sorts are selecting themselves out of the population? And with negative fertility rates + unprecedented levels of immigration, how smart are our immigrants (and their children?)

Data on these questions is not the easiest to find. Jayman has data on African American fertility (dysgenic,) but white American fertility may be currently eugenic (after several decades of dysgenics.) Jayman also notes a peculiar gender difference in these trends: female fertility is strongly dysgenic, while male is eugenic (for both whites and blacks). Given that historically, about 80% of women reproduced vs. only 40% of males, I think it likely that this pattern has always been true: women only want to marry intelligent, high-performing males, while males are okay with marrying dumb women. (Note: the female ability to detect intelligence may be broken by modern society.)

Counter-Currents has a review of Lynn’s Dysgenics with some less hopeful statistics, like an estimation that Greece lost 5 IQ points during the Baby Boom, which would account for their current economic woes. (Overall, I think the Baby Boom had some definite negative effects on the gene pool that are now working their way out.)

Richwine estimates the IQ of our immigrant Hispanic-American population at 89.2, with a slight increase for second and third-generation kids raised here. Since the average American IQ is 98 and Hispanics are our fastest-growing ethnic group, this is strongly dysgenic. (The rest of our immigrants, from countries like China, are likely to be higher-IQ than Americans.) However, since Hispanic labor is typically used to avoid African American (reported 85 average IQ) labor, the replacement of African Americans with Mexicans is locally eugenic–hence the demand for Hispanic labor.

Without better data, none of this conclusively proves whether fertility in the West is currently eugenic or dysgenic, but I can propose three main factors that should be watched for their potentially negative effects:

  1. Immigration (obviously.)
  2. Welfare–I suspect the greater black reliance on welfare may be diving black dysgenics, but some other factor like crime could actually be at play.
  3. Anti-child culture.

I’m going to focus on the last one because it’s the only one that hasn’t already been explained in great detail elsewhere.

For American women, childbearing is low-class and isolating.

For all our fancy talk about maternity leave, supporting working moms, etc., America is not a child-friendly place. Society frowns on loud, rambunctious children running around in public, and don’t get me started on how public schools deal with boys. Just try to find something entertaining for both kids and grown-ups that doesn’t cost an arm and a leg for larger families–admission to the local zoo for my family costs over $50 and requires over an hour, round trip, of driving. (And it isn’t even a very good zoo.) Now try to find an activity your childless friends would also like to do with you.

Young women are constantly told that getting pregnant will ruin their lives (most vocally by their own parents,) and that if they want to stay home and raise children, they are social parasites. (Yes, literally.) We see child-rearing, like tomato picking, as a task best performed by low-wage immigrant daycare workers.

I am reminded here of a mom’s essay I read about the difference in attitudes toward children in the US and Israel, the only Western nation with a positive native fertility rate. Israel, as she put it, is a place where children are valued and “kids can be kids.” I’ve never been to Israel, so I’ll just have to trust her:

How Israelis love kids, anyone’s kids. The country is a free-for-all for the youngest set, something I truly appreciated only once I started bringing my own children there. When I was a teenager visiting Israel from the States, I noticed how people there just don’t allow a child to cry. One pout, one sob, and out comes candy, trinkets and eager smiles to turn a kid around. That would never happen back home—a stranger give a child candy?!—but in Israel, in a nation that still harbors a post-Holocaust mentality, there is no reason that a Jewish child should ever cry again, if someone can help it.

Incidentally, if you qualify under Israeli health care law, you can get a free, state-funded abortion. Abortion doesn’t appear to have destroyed Israel’s fertility.

Since male fertility is (probably) already eugenic, then the obvious place to focus is female fertility: make your country a place where children are actively valued and intelligent women are encouraged instead of insulted for wanting them, and–hopefully–things can improve.

In Defense of Planned Parenthood

Abortion and birth control are important tools in the ultimate human thriving toolkit.

Unless you want to eliminate all the robots and go back to agricultural labor (which is not going to get you an interstellar society,) you will have to deal, somehow, with all of the humans who don’t have the chops to survive in society. Letting people starve in the streets is inhumane and inspires people to fund large social welfare states, which may have negative long-term effects.

Historically, death rates were very high, especially infant mortality. My great-great grandparents lost over half of their 16 children before the age of five; such was normal.

The effects of declining infant mortality are happy parents, of course, but also long-term degradation of the gene pool, overpopulation, and eventual systemic collapse as we burn through the Earth’s resources. We’re already seeing this, both in increasing reaction times (it looks like Whites are getting dumber, and Ashkenazi IQ is probably plummeting, relatively speaking,) and the flooding of high-breeding peoples out of their exhausted biomes into fresh territory to consume (to the detriment of those trying to maintain a non-degraded biome.)

As I believe I have mentioned before, there is nothing like a parenting forum to convince you that parents are idiots. Unfortunately, a very large percentage of people become parents because they are too dumb not to.

I recently had a conversation with a friend who tried to reassure me that this was not a problem. “Don’t worry,” they said. “Dumb people have always had more kids than smart people.”

“No,” I said. “No no no. Dumb people did not historically have more kids than smart people.” History was brutal; 20-50% infant mortality was the norm, and people who did a better job taking care of and providing for their children had more children who made it to adulthood than those who didn’t.

No one in their right mind wants to simply eliminate all maternal and childhood medical care (and hygiene) so we can return to the age of high infant mortality. There are far better solutions than giving everyone Smallpox and seeing who makes it. But you also do not want a situation where the primary barrier to reproduction is actually intelligence.

The obvious solution is free IUDs for everyone. Globally. The long-term planners will get theirs removed when they’re ready to have children, and the short term planners will be able to go about their business without making “oopsies.” People who want 18 children will still be able to have 18 children, but people who don’t have the resources to support children don’t have to have any.

Abortion also plays an important role in the maintenance of modern society. Ideally, free abundant birth control would eliminate most of the need for abortion, but there will always be mistakes, medical complications, and non-viable fetuses of various sorts. Eliminate these earlier, not later.

These are not the children of intelligent, healthy, well-adjusted people who have some weird phobia of childbirth. These are fetuses with health problems and fetuses whose parents don’t have the resources, mentally or physically, to take care of them. The apple does not fall far from the tree, and genetically, those children will inherit their parents’ traits. If you are not volunteering to raise those fetuses (and their fetuses) yourself, then I think you should give some serious thought to who you think will.

After all is said and done, I don’t care what Planned Parenthood does with aborted fetuses, so long as they’re disposed of hygienically. They’re already dead, for goodness sakes.